%0 Journal Article %J Prevention Science %D 2024 %T Family-focused universal substance use prevention in primary care: Advancing a pragmatic national healthcare agenda %A Hogue, Aaron %A Brykman, Kelsey %A Guilamo-Ramos, Vincent %A Ilakkuvan, Vinu %A Kuklinski, Margaret R. %A Matson, Pamela %A McKnight, Erin R. %A Powell, Terrinieka W. %A Richter, Linda %A Walker-Harding, Leslie R. %B Prevention Science %V 25 %P 307-317 %8 02/2024 %G eng %N 2 %R 10.1007/s11121-023-01584-4 %0 Journal Article %J Prevention Science %D 2022 %T State-of-the-art in substance use prevention and early intervention: Applications to pediatric primary care settings %A Matson, Pamela A. %A Ridenour, Ty %A Ialongo, Nicholas %A Spoth, Richard %A Prado, Guillermo %A Hammond, Christopher J. %A Hawkins, J D %A Adger, Hoover, Jr. %B Prevention Science %V 23 %P 204-211 %G eng %0 Journal Article %J Development and Psychopathology %D 2017 %T Time-varying effects of families and peers on adolescent marijuana use: Person-environment interactions across development %A Epstein, Marina %A Hill, Karl G. %A Roe, Stephanie S. %A Bailey, Jennifer A. %A Iacono, William G. %A McGue, Matt %A Kristman-Valente, Allison %A Catalano, Richard F. %A Haggerty, Kevin P. %B Development and Psychopathology %V 29 %P 887-900 %G eng %0 Journal Article %J Behavior Genetics %D 2016 %T A test-replicate approach to candidate gene research on addiction and externalizing disorders: A collaboration across five longitudinal studies %A Samek, Diana R. %A Bailey, Jennifer A. %A Hill, Karl G. %A Wilson, Sylia %A Lee, Susanne %A Keyes, Margaret A. %A Epstein, Marina %A Smolen, Andrew %A Miller, Michael %A Winters, Ken C. %A Hawkins, J D %A Catalano, Richard F. %A Iacono, William G. %A McGue, Matt %B Behavior Genetics %I Springer %V 46 %P 608-626 %G eng %0 Journal Article %J Dev Psychopathol %D 2016 %T Time-varying effects of families and peers on adolescent marijuana use: Person-environment interactions across development. %A Epstein, Marina %A Hill, Karl G %A Roe, Stephanie S %A Bailey, Jennifer A %A Iacono, William G %A McGue, Matt %A Kristman-Valente, Allison %A Catalano, Richard F %A Haggerty, Kevin P %X

Studies have demonstrated that the effects of two well-known predictors of adolescent substance use, family monitoring and antisocial peers, are not static but change over the course of adolescence. Moreover, these effects may differ for different groups of youth. The current study uses time-varying effect modeling to examine the changes in the association between family monitoring and antisocial peers and marijuana use from ages 11 to 19, and to compare these associations by gender and levels of behavioral disinhibition. Data are drawn from the Raising Healthy Children study, a longitudinal panel of 1,040 youth. The strength of association between family monitoring and antisocial peers and marijuana use was mostly steady over adolescence, and was greater for girls than for boys. Differences in the strength of the association were also evident by levels of behavioral disinhibition: youth with lower levels of disinhibition were more susceptible to the influence of parents and peers. Stronger influence of family monitoring on girls and less disinhibited youth was most evident in middle adolescence, whereas the stronger effect of antisocial peers was significant during middle and late adolescence. Implications for the timing and targeting of marijuana preventive interventions are discussed.

%B Dev Psychopathol %P 1-14 %8 2016 Jul 15 %G ENG %1 http://www.ncbi.nlm.nih.gov/pubmed/27417425?dopt=Abstract %R 10.1017/S0954579416000559 %0 Journal Article %J Drug Alcohol Depend %D 2014 %T General and substance-specific predictors of young adult nicotine dependence, alcohol use disorder, and problem behavior: replication in two samples. %A Bailey, J A %A Samek, D R %A Keyes, M A %A Hill, K G %A Hicks, B M %A McGue, M %A Iacono, W G %A Epstein, M %A Catalano, R F %A Haggerty, K P %A Hawkins, J D %K Adolescent %K Adult %K Alcohol-Related Disorders %K Antisocial Personality Disorder %K Child %K Family Health %K Female %K Humans %K Male %K Minnesota %K Northwestern United States %K Risk Factors %K Substance-Related Disorders %K Tobacco Use Disorder %K Twins %K Unsafe Sex %K Young Adult %X

BACKGROUND: This paper presents two replications of a heuristic model for measuring environment in studies of gene-environment interplay in the etiology of young adult problem behaviors.

METHODS: Data were drawn from two longitudinal, U.S. studies of the etiology of substance use and related behaviors: the Raising Healthy Children study (RHC; N=1040, 47% female) and the Minnesota Twin Family Study (MTFS; N=1512, 50% female). RHC included a Pacific Northwest, school-based, community sample. MTFS included twins identified from state birth records in Minnesota. Both studies included commensurate measures of general family environment and family substance-specific environments in adolescence (RHC ages 10-18; MTFS age 18), as well as young adult nicotine dependence, alcohol and illicit drug use disorders, HIV sexual risk behavior, and antisocial behavior (RHC ages 24, 25; MTFS age 25).

RESULTS: Results from the two samples were highly consistent and largely supported the heuristic model proposed by Bailey et al. (2011). Adolescent general family environment, family smoking environment, and family drinking environment predicted shared variance in problem behaviors in young adulthood. Family smoking environment predicted unique variance in young adult nicotine dependence. Family drinking environment did not appear to predict unique variance in young adult alcohol use disorder.

CONCLUSIONS: Organizing environmental predictors and outcomes into general and substance-specific measures provides a useful way forward in modeling complex environments and phenotypes. Results suggest that programs aimed at preventing young adult problem behaviors should target general family environment and family smoking and drinking environments in adolescence.

%B Drug Alcohol Depend %V 138 %P 161-8 %8 2014 May 1 %G eng %R 10.1016/j.drugalcdep.2014.02.023 %0 Journal Article %J Lancet %D 2012 %T Worldwide application of prevention science in adolescent health. %A Catalano, Richard F %A Fagan, Abigail A %A Gavin, Loretta E %A Greenberg, Mark T %A Irwin, Charles E %A Ross, David A %A Shek, Daniel T L %K Adolescent Medicine %K Child %K Global Health %K Government Programs %K Health Behavior %K Health Status %K Humans %K Public Health %K Young Adult %X

The burden of morbidity and mortality from non-communicable disease has risen worldwide and is accelerating in low-income and middle-income countries, whereas the burden from infectious diseases has declined. Since this transition, the prevention of non-communicable disease as well as communicable disease causes of adolescent mortality has risen in importance. Problem behaviours that increase the short-term or long-term likelihood of morbidity and mortality, including alcohol, tobacco, and other drug misuse, mental health problems, unsafe sex, risky and unsafe driving, and violence are largely preventable. In the past 30 years new discoveries have led to prevention science being established as a discipline designed to mitigate these problem behaviours. Longitudinal studies have provided an understanding of risk and protective factors across the life course for many of these problem behaviours. Risks cluster across development to produce early accumulation of risk in childhood and more pervasive risk in adolescence. This understanding has led to the construction of developmentally appropriate prevention policies and programmes that have shown short-term and long-term reductions in these adolescent problem behaviours. We describe the principles of prevention science, provide examples of efficacious preventive interventions, describe challenges and potential solutions to take efficacious prevention policies and programmes to scale, and conclude with recommendations to reduce the burden of adolescent mortality and morbidity worldwide through preventive intervention.

%B Lancet %V 379 %P 1653-64 %8 2012 Apr 28 %G eng %N 9826 %R 10.1016/S0140-6736(12)60238-4 %0 Journal Article %J Arch Gen Psychiatry %D 2010 %T A meta-analysis of depression during pregnancy and the risk of preterm birth, low birth weight, and intrauterine growth restriction. %A Grote, Nancy K %A Bridge, Jeffrey A %A Gavin, Amelia R %A Melville, Jennifer L %A Iyengar, Satish %A Katon, Wayne J %K Cross-Cultural Comparison %K Depressive Disorder %K Female %K Fetal Growth Retardation %K Humans %K Infant, Low Birth Weight %K Infant, Newborn %K Mass Screening %K Obstetric Labor, Premature %K Pregnancy %K Pregnancy Complications %K Prospective Studies %K Risk %K Socioeconomic Factors %X

CONTEXT: Maternal depressive symptoms during pregnancy have been reported in some, but not all, studies to be associated with an increased risk of preterm birth (PTB), low birth weight (LBW), and intrauterine growth restriction (IUGR).

OBJECTIVE: To estimate the risk of PTB, LBW, and IUGR associated with antenatal depression.

DATA SOURCES AND STUDY SELECTION: We searched for English-language and non-English-language articles via the MEDLINE, PsycINFO, CINAHL, Social Work Abstracts, Social Services Abstracts, and Dissertation Abstracts International databases (January 1980 through December 2009). We aimed to include prospective studies reporting data on antenatal depression and at least 1 adverse birth outcome: PTB (<37 weeks' gestation), LBW (<2500 g), or IUGR (<10th percentile for gestational age). Of 862 reviewed studies, 29 US-published and non-US-published studies met the selection criteria.

DATA EXTRACTION: Information was extracted on study characteristics, antenatal depression measurement, and other biopsychosocial risk factors and was reviewed twice to minimize error.

DATA SYNTHESIS: Pooled relative risks (RRs) for the effect of antenatal depression on each birth outcome were calculated using random-effects methods. In studies of PTB, LBW, and IUGR that used a categorical depression measure, pooled effect sizes were significantly larger (pooled RR [95% confidence interval] = 1.39 [1.19-1.61], 1.49 [1.25-1.77], and 1.45 [1.05-2.02], respectively) compared with studies that used a continuous depression measure (1.03 [1.00-1.06], 1.04 [0.99-1.09], and 1.02 [1.00-1.04], respectively). The estimates of risk for categorically defined antenatal depression and PTB and LBW remained significant when the trim-and-fill procedure was used to correct for publication bias. The risk of LBW associated with antenatal depression was significantly larger in developing countries (RR = 2.05; 95% confidence interval, 1.43-2.93) compared with the United States (RR = 1.10; 95% confidence interval, 1.01-1.21) or European social democracies (RR = 1.16; 95% confidence interval, 0.92-1.47). Categorically defined antenatal depression tended to be associated with an increased risk of PTB among women of lower socioeconomic status in the United States.

CONCLUSIONS: Women with depression during pregnancy are at increased risk for PTB and LBW, although the magnitude of the effect varies as a function of depression measurement, country location, and US socioeconomic status. An important implication of these findings is that antenatal depression should be identified through universal screening and treated.

%B Arch Gen Psychiatry %V 67 %P 1012-24 %8 2010 Oct %G eng %N 10 %R 10.1001/archgenpsychiatry.2010.111 %0 Journal Article %J Public Health Rep %D 2010 %T Risk factors for HIV disease progression in a rural southwest American Indian population. %A Iralu, Jonathan %A Duran, Bonnie %A Pearson, Cynthia R %A Jiang, Yizhou %A Foley, Kevin %A Harrison, Melvin %K Alcoholism %K CD4 Lymphocyte Count %K Disease Progression %K Female %K HIV Infections %K Humans %K Indians, North American %K Male %K Medication Adherence %K Medicine, Traditional %K Prisoners %K Risk Factors %K Rural Population %K Southwestern United States %K Viral Load %X

OBJECTIVES: Risk factors for human immunodeficiency virus (HIV) disease progression among American Indians (AIs) have been poorly characterized. We assessed the impact of socioeconomic factors and use of traditional healing on HIV disease progression in a rural AI community.

METHODS: From January 2004 through December 2006, we interviewed 36 HIV-positive AIs regarding their socioeconomic status, incarceration, and use of traditional healing. We also collected chart-abstracted adherence and substance-abuse data. Through bivariate analysis, we compared these factors with the CD4-cell counts and log HIV-1 viral loads (VLs). Using a simple regression model, we assessed interactions between the significant associations and the outcome.

RESULTS: Participant characteristics included being male (58.3%), being transgender (13.9%), having ever been incarcerated (63.9%), having a household income of < $1,000/month (41.7%), being unemployed (61.1%), being diagnosed with alcohol abuse (50.0%), and using traditional medicine (27.8%) in the last 12 months. Higher VLs were associated with recent incarceration (p < 0.05), household income of < $1,000/month (p < 0.05), and provider-assessed alcohol abuse (p < 0.05). We found an interaction between incarceration and alcohol abuse, and alcohol abuse was the factor more strongly associated with higher VLs. A lower CD4 count was associated with recent incarceration (p < 0.05) and use of traditional medicine (p < 0.05).

CONCLUSIONS: Alcohol abuse is an important contributor to HIV disease progression, and participants with lower CD4 counts were more likely to use traditional medicine. HIV care among this rural AI population should focus on addressing alcohol abuse and other socioeconomic risk factors and promote collaboration between Western medical and Navajo traditional practitioners.

%B Public Health Rep %V 125 Suppl 4 %P 43-50 %8 2010 Jul-Aug %G eng